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ct26 murine colorectal carcinoma cell line  (ATCC)


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    ATCC ct26 murine colorectal carcinoma cell line
    Combination RT and anti-CCR8 reduces tumor burden and improve survival in distant non-irradiated tumors BALB/c mice were injected with <t>CT26</t> on both flanks. Tumor-bearing mice were treated with a “pre” treatment strategy. Mice were injected i.p. with anti-CCR8 on days 7, 10, and 14 and treated with 12 Gy RT to the right flank tumor (treated) on day14 post-tumor inoculation. The left tumor (untreated) did not receive RT. Tumor growth and survival were assessed. A total of 8 mice were analyzed per group. Data are represented as mean ± SD. Statistics for survival plots were performed using a Mantel-Cox test between groups. ns = not significant, ∗∗∗∗ p < 0.0001, ∗∗∗ p < 0.001, ∗∗ p < 0.01, ∗ p < 0.05.
    Ct26 Murine Colorectal Carcinoma Cell Line, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 3501 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/ct26 murine colorectal carcinoma cell line/product/ATCC
    Average 99 stars, based on 3501 article reviews
    ct26 murine colorectal carcinoma cell line - by Bioz Stars, 2026-03
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    1) Product Images from "Fluorescence tracking Treg movement identifies anti-CCR8 and radiation as a therapeutic combination"

    Article Title: Fluorescence tracking Treg movement identifies anti-CCR8 and radiation as a therapeutic combination

    Journal: iScience

    doi: 10.1016/j.isci.2025.114572

    Combination RT and anti-CCR8 reduces tumor burden and improve survival in distant non-irradiated tumors BALB/c mice were injected with CT26 on both flanks. Tumor-bearing mice were treated with a “pre” treatment strategy. Mice were injected i.p. with anti-CCR8 on days 7, 10, and 14 and treated with 12 Gy RT to the right flank tumor (treated) on day14 post-tumor inoculation. The left tumor (untreated) did not receive RT. Tumor growth and survival were assessed. A total of 8 mice were analyzed per group. Data are represented as mean ± SD. Statistics for survival plots were performed using a Mantel-Cox test between groups. ns = not significant, ∗∗∗∗ p < 0.0001, ∗∗∗ p < 0.001, ∗∗ p < 0.01, ∗ p < 0.05.
    Figure Legend Snippet: Combination RT and anti-CCR8 reduces tumor burden and improve survival in distant non-irradiated tumors BALB/c mice were injected with CT26 on both flanks. Tumor-bearing mice were treated with a “pre” treatment strategy. Mice were injected i.p. with anti-CCR8 on days 7, 10, and 14 and treated with 12 Gy RT to the right flank tumor (treated) on day14 post-tumor inoculation. The left tumor (untreated) did not receive RT. Tumor growth and survival were assessed. A total of 8 mice were analyzed per group. Data are represented as mean ± SD. Statistics for survival plots were performed using a Mantel-Cox test between groups. ns = not significant, ∗∗∗∗ p < 0.0001, ∗∗∗ p < 0.001, ∗∗ p < 0.01, ∗ p < 0.05.

    Techniques Used: Irradiation, Injection



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    Image Search Results


    Combination RT and anti-CCR8 reduces tumor burden and improve survival in distant non-irradiated tumors BALB/c mice were injected with CT26 on both flanks. Tumor-bearing mice were treated with a “pre” treatment strategy. Mice were injected i.p. with anti-CCR8 on days 7, 10, and 14 and treated with 12 Gy RT to the right flank tumor (treated) on day14 post-tumor inoculation. The left tumor (untreated) did not receive RT. Tumor growth and survival were assessed. A total of 8 mice were analyzed per group. Data are represented as mean ± SD. Statistics for survival plots were performed using a Mantel-Cox test between groups. ns = not significant, ∗∗∗∗ p < 0.0001, ∗∗∗ p < 0.001, ∗∗ p < 0.01, ∗ p < 0.05.

    Journal: iScience

    Article Title: Fluorescence tracking Treg movement identifies anti-CCR8 and radiation as a therapeutic combination

    doi: 10.1016/j.isci.2025.114572

    Figure Lengend Snippet: Combination RT and anti-CCR8 reduces tumor burden and improve survival in distant non-irradiated tumors BALB/c mice were injected with CT26 on both flanks. Tumor-bearing mice were treated with a “pre” treatment strategy. Mice were injected i.p. with anti-CCR8 on days 7, 10, and 14 and treated with 12 Gy RT to the right flank tumor (treated) on day14 post-tumor inoculation. The left tumor (untreated) did not receive RT. Tumor growth and survival were assessed. A total of 8 mice were analyzed per group. Data are represented as mean ± SD. Statistics for survival plots were performed using a Mantel-Cox test between groups. ns = not significant, ∗∗∗∗ p < 0.0001, ∗∗∗ p < 0.001, ∗∗ p < 0.01, ∗ p < 0.05.

    Article Snippet: The CT26 murine colorectal carcinoma cell line was purchased from ATCC (CRL-2638).

    Techniques: Irradiation, Injection

    Fusobacte rium nucleatum ( Fn ) promotes colorectal cancer metastasis. Polymerase chain reaction analysis revealed an increased abundance of Fn in the feces of patients with colorectal cancer, with a significantly greater abundance in patients with lymph node metastasis than in those without metastasis (A and B). A total of 45 pairs of colorectal cancer tissues and adjacent tissues were collected. Quantitative polymerase chain reaction analysis of C. difficile abundance in cancer tissues revealed a significant increase compared to that in adjacent tissues, with even greater abundance in situ cancer tissues from patients with lymph node metastasis (C and D). Fluorescence in situ hybridization experiments detected C. difficile in the lymph node metastases of colorectal cancer patients (E and F). Coculture of colorectal cancer cells (HCT-116 and LoVo) with C. difficile significantly promoted the in vitro migration of cancer cells, as shown by Transwell migration assays (G and H) and scratch assays (I). In a mouse model of colorectal cancer lung metastasis established via tail vein injection, coculture of colorectal cancer cells significantly increased the number and size of metastatic foci in the lung (J and K). In a mouse model of colorectal cancer liver metastasis established via splenic injection, the coculture group showed a significantly increased number of liver metastatic foci (L).

    Journal: The Turkish Journal of Gastroenterology

    Article Title: Gut Microbiome Diagnostic Biomarkers for Colorectal Cancer

    doi: 10.5152/tjg.2025.24810

    Figure Lengend Snippet: Fusobacte rium nucleatum ( Fn ) promotes colorectal cancer metastasis. Polymerase chain reaction analysis revealed an increased abundance of Fn in the feces of patients with colorectal cancer, with a significantly greater abundance in patients with lymph node metastasis than in those without metastasis (A and B). A total of 45 pairs of colorectal cancer tissues and adjacent tissues were collected. Quantitative polymerase chain reaction analysis of C. difficile abundance in cancer tissues revealed a significant increase compared to that in adjacent tissues, with even greater abundance in situ cancer tissues from patients with lymph node metastasis (C and D). Fluorescence in situ hybridization experiments detected C. difficile in the lymph node metastases of colorectal cancer patients (E and F). Coculture of colorectal cancer cells (HCT-116 and LoVo) with C. difficile significantly promoted the in vitro migration of cancer cells, as shown by Transwell migration assays (G and H) and scratch assays (I). In a mouse model of colorectal cancer lung metastasis established via tail vein injection, coculture of colorectal cancer cells significantly increased the number and size of metastatic foci in the lung (J and K). In a mouse model of colorectal cancer liver metastasis established via splenic injection, the coculture group showed a significantly increased number of liver metastatic foci (L).

    Article Snippet: The human colorectal carcinoma cells (HCT-116) and LoVo were obtained from the American Type Culture Collection (ATCC).

    Techniques: Polymerase Chain Reaction, Real-time Polymerase Chain Reaction, In Situ, Fluorescence, In Situ Hybridization, In Vitro, Migration, Injection